CORDIS Project
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This project explores cardiac fibrosis and regeneration following myocardial infarction using a zebrafish model. It aims to understand the mechanisms of myofibroblast function and extracellular matrix regression to enhance cardiac repair and identify potential therapeutic targets.
Myocardial infarction (MI) leads to cardiomyocyte death and accumulation of myofibroblasts (MFs) at the site of injury, which produce large amounts of extracellular matrix (ECM), generating a scar.
Initially, cardiac fibrosis protects from ventricular wall rupture, but subsequent myocardial remodelling causes heart failure, representing a leading cause of death in Europe.
While MFs play a central role in cardiac fibrosis, there is confusion on their origin, a lack of specific markers and the exi…
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