CORDIS Project
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This project investigates how the vaccinia virus interacts with the host cell's degradation machinery during its replication cycle. It aims to uncover the mechanisms by which the virus manipulates cellular processes to facilitate its lifecycle and may provide insights into cellular regulation.
Vaccinia virus (VACV), the prototypic poxvirus, is a large, enveloped, DNA virus characterized by its cytoplasmic site of replication and large subset of genes.
Due to the complexity of VACV, the majority of studies focus on the virus rather than the host cell.
Thus the repertoire of cell factors and functions required for its replication remain largely unknown.
Our previous work to define a subset of these, revealed the cellular degradation machinery as a key requirement of VACV replication.
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THE UNIVERSITY OF BIRMINGHAM
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United Kingdom, London
Type: University / higher education
Activity type: Higher or Secondary Education Establishments
SME: No
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