CORDIS Project
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This project explores the role of autophagy in the turnover of synaptic proteins, which are crucial for neuronal communication. By employing various biological techniques, it aims to clarify how autophagy influences synaptic structure and function, contributing to our understanding of brain health and disease.
Neuronal communication primarily occurs at synapses.
Recent studies have suggested a key role for protein synthesis and degradation in the regulation of synaptic structure, function and plasticity by changing the abundance of select synaptic proteins in a spatially confined manner.
Remodeling of the synaptic proteome is accomplished by, for example, regulated degradation via the ubiquitin proteasome system or by autophagy.
Autophagy has been shown to modulate synaptic vesicle numbers and neurotr…
Partner organizations (coordinator is shown above), with normalized type and CORDIS activity type. Guests see up to 4 partners.
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