CORDIS Project
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This project investigates how fibroblasts behave after muscle injury, particularly in Duchenne Muscular Dystrophy. It aims to understand the differences in fibroblast populations and their roles in muscle regeneration versus fibrosis, potentially leading to personalized treatment strategies.
Following skeletal muscle injury, resident fibroblasts proliferate and release cytokines that enhance muscle regeneration.
Fibroblasts also secrete chemokines to recruit cellular migration, and extracellular proteins that stabilize the site of injury.
However, in chronic diseases, such as Duchenne Muscular Dystrophy (DMD), continual muscle damage shifts fibroblasts towards an uncontrolled fibrotic and inflammatory phenotype.
Excessive fibrosis and inflammation are prominent pathological features…
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