CORDIS Project
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This project investigates the regulation of the SCN5A gene, which is crucial for cardiac sodium channel function. By exploring transcription factors, DNA methylation, and microRNA interactions, the research aims to enhance understanding of cardiac arrhythmias and sudden cardiac death syndromes.
Sudden cardiac death syndromes are an important cause of mortality in young individuals in developed countries.
The SCN5A gene, which encodes the alpha subunit of the cardiac sodium channel, plays a key role in controlling the sodium currents during the action potential.
When malfunctioning due to loss-of-function genetic defects, SCN5A may be responsible for lethal diseases like Brugada syndrome or familial conduction disease.
However, little is known regarding SCN5A gene expression regulation,…
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