CORDIS Project
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This project investigates how HIV-1 infection is controlled at the gene expression level in patients receiving antiretroviral therapy and in elite controllers. It aims to identify host factors and molecular mechanisms that enable effective control of viral replication, which could inform future treatment strategies.
Human Immunodeficiency virus type 1 (HIV-1), the causative agent of AIDS, infects primarily cells of the immune system.
The outcome of HIV-1 infection results from complex interactions between viral proteins and host cell factors (ref).
In most cases, HIV-1 successfully hijacks cellular pathways and bypasses cellular restriction factors for optimal replication leading to continuous rounds of infection, replication and cell death (ref).
Continuous viral replication causes the loss of CD4+T cells…
CENTRE NATIONAL DE LA RECHERCHE SCIENTIFIQUE CNRS
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