CORDIS Project
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This project explores the role of Calcium-calmodulin-dependent protein kinase II (CaMKII) in memory formation and storage in neurons. By creating light-controllable variants of CaMKII, the research aims to clarify its involvement in long-term potentiation and other neuronal functions.
How are memories formed and stored in the brain?
One candidate mechanism is long term potentiation (LTP), a form of synaptic plasticity that is characterized by strengthening of synaptic connections between neurons in the brain, lasting from minutes to hours.
The first step in LTP is a sharp influx of calcium ions, which serves to activate many Ca2+-dependent proteins, one of which is Calcium-calmodulin-dependent protein kinase II (CaMKII).
Once activated, CaMKII translocates to the post-synapse…
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