CORDIS Project
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This project investigates the molecular causes of cancer cachexia, a condition leading to muscle loss in cancer patients. It focuses on the role of LRPPRC, a protein involved in mitochondrial function, and aims to explore its potential as a therapeutic target to improve patient outcomes.
Cancer cachexia (CC) affects 50-80% of patients with cancer and is characterized by accelerated loss of muscle mass, which is directly linked to reduced quality of life, treatment toxicity, and lower survival.
With no pharmacological treatment options, key to improving life expectancy is to understand the molecular determinants of CC, however, these are poorly defined.
Mitochondrial dysfunction and inflammation can cause muscle atrophy and are considered as emerging hallmarks of CC.
Exercise is…
KOBENHAVNS UNIVERSITET
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