CORDIS Project
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This research investigates how the malaria parasite Plasmodium falciparum evades the human immune system through antigenic variation. By understanding the genetic mechanisms behind this process, the project aims to identify new strategies for drug and vaccine development to combat malaria.
The deadliest form of human malaria is caused by the protozoan parasite, Plasmodium falciparum, which annually infects millions worldwide.
Its virulence is attributed to its ability to evade the human immune system, by modifying the host red blood cell surface to adhere to the vascular endothelium and to undergo antigenic variation.
Antigenic variation is achieved through switches in expression of hypervariable surface ligands named PfEMP1.
These proteins are encoded by a multi-copy gene family…
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