CORDIS Project
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This research explores the regulation of gene expression in the malaria parasite Plasmodium falciparum, focusing on how it evades the human immune system. By understanding the mechanisms behind gene activation, the project aims to identify new targets for malaria treatment and vaccine development.
The deadliest form of human malaria is caused by the protozoan parasite, Plasmodium falciparum, which annually infects millions worldwide.
The virulence of P. falciparum is attributed to its ability to evade the human immune system, by modifying the host red blood cell surface to adhere to the vascular endothelium and to undergo antigenic variation.
The main cell surface ligands responsible for both cytoadherence and antigenic variation are members of the P. falciparum Erythrocyte Membrane Prote…
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