CORDIS Project
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This project aims to understand the mechanisms behind insulin secretion failures in diabetes, particularly focusing on ATP-sensitive potassium channels. It seeks to improve treatment options for neonatal diabetes and explore the neurological implications of severe mutations.
We are currently experiencing a fast-growing diabetes pandemic.
Both type 2 diabetes and rare monogenic forms of diabetes, such as neonatal diabetes, are characterised by impaired insulin secretion.
This project seeks to resolve the fundamental mechanisms underlying insulin secretion and its failure in diabetes.
We have shown that activating mutations in the ATP-sensitive potassium (KATP) channel cause neonatal diabetes, which has enabled children with this disease to switch from insulin injecti…
THE CHANCELLOR, MASTERS AND SCHOLARS OF THE UNIVERSITY OF OXFORD
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