CORDIS Project
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This project investigates the role of formin proteins in the actin-based movement of apicomplexan parasites like Plasmodium, which causes malaria. The research aims to characterize the structural and biochemical properties of these proteins to better understand their function in parasite motility.
Funding is applied to carry out research on formin-mediated regulation and dynamics of actin-based motility of apicomplexan parasites, such as Plasmodium, the causative agent of malaria.
Formins are large multi-domain proteins, which regulate microfilament formation by triggering actin monomer addition from actin-profilin complexes at the barbed end.
Formins bear two formin homology (FH) domains.
Plasmodium FH2 domains are conserved with higher eukaryotes and are responsible for actin binding, w…
OULUN YLIOPISTO
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