CORDIS Project
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This project investigates how mutations in the KRAS oncogene influence glycosylation and glycan-binding proteins in pancreatic ductal adenocarcinoma. By understanding these interactions, the research aims to identify new therapeutic targets to combat this aggressive cancer.
Pancreatic ductal adenocarcinoma (PDAC) stands out as one of the most aggressive tumors, ranking as the third leading cause of cancer-related death.
Glycosylation has emerged as a critical regulatory mechanism contributing to tumor progression and immunosuppression, a hallmark of PDAC tumor microenvironment (TME).
Recent studies have shown that tumor immune evasion programs can be facilitated by the activation of immunosuppressive glyco-immune checkpoints (GICs), governed by interactions between…
AGENCIA ESTATAL CONSEJO SUPERIOR DE INVESTIGACIONES CIENTIFICAS
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